oong a stuaiglit Tine The remainder of the examination was n

oong a stuaiglit Tine. The remainder of the examination was normal. Case 4 Presentation: A 15-year old male patient was admitted to the ER because an inability to swallow his breakfast. He had been discharged from the hospital 36-hours prior following two weeks of recovery from a motor vehicle accident that resulted in a compound tib-fib fracture, as well as other broken bones. Neurological Exam: Touch discrimination of the left forehead is impaired and mild temperature sensation in the right hand is absent. Gag reflex is not present upon tactile stimulation of the soft palate. No other motor, sensory, or behavioral findings were present in the neurological exanm. History: The patient had been previously diagnosed (at 12 years of age) with an atrial septal defect (ASD) of the heart. No family history is of consequence to this case.

Solution

(1)There are numerous reasons of dysphagia. Some narrate to glitches with the gullet – the brawny passageway linking the entrance to the abdominal. Such glitches could be connected to dysfunction of the esophageal strengths, growths in the gullet, incomplete obstruction of the throat, injury to esophageal soft tissue from stomach acid, and others. Other reasons are connected to flagging of the throat muscles that may occur due to nervous complaints such as manifold sclerosis, blow, or brain wound. Also existence alike in name, the two complaints are really fairly separate in their countryside. Aphasia is a language and verbal complaint that consequences from nervous injury to parts complicated in linguistic dispensation. Dysphagia is not connected to language or linguistic manufacture or dispensation. A complaint that often co-occurs with aphasia – apraxia is more alike to dysphasia in that it also contains the musculature of the articulators. Though, apraxia is also not distinct as problems swallowing but to construction of speech noises.

(2) Patients with an irregular right subclavian artery (ARSA) and leftward aortic arch (LAA) could have related cardiac irregularities. Patients with this irregularity are typically asymptomatic. Even nevertheless this illness is a congenital irregularity, patients typically do not current with it in infantile. They develop as indicative in early maturity and even in central or old stage. Models amplification in this late demonstration comprise physical and functional variations that could happen with the mature procedure, such as, augmented esophageal inflexibility, inflexibility of the container partition due to atherosclerosis, elongation of the aorta, besides aortic aneurysm creation, particularly in the company of diverticulum.