What is the main cell structure that is affected by multiple

What is the main cell structure that is affected by multiple sclerosis and how is the action potential of a neuron affected by this disease
What is the main cell structure that is affected by multiple sclerosis and how is the action potential of a neuron affected by this disease

Solution

Multiple sclerosis is the demyelinating disorder in which “myelin of neurons” broken due to the various factors in both neonatal and child brain finally leading severe cognitive and sensory –motor impairment due to the improper functioning of the flexion reflex. Potential medication and associated treatment is very difficult as the specific drug molecules, which can act against this gene coding proteins for the synthesis of myelin.

During multiple sclerosis, merely central nervous system with oligodendrocytes will be affected thereby improper myelination around the CNS neurons. Schwann cells will not get affected because these are myelin-producing cells in peripheral nervous system. This disease is pertaining to central nervous system is leading to complete paralysis of the muscle tone innervated with motor neurons during the functioning of the flexion reflex on the specific skeletal muscle result in irreversible flaccid & paralysis with no muscle tone. A peripheral nervous system problem can be irreversible as it can regenerate the activity of spastic nature of the muscle. Multiple sclerosis is an autoimmune disease in which, the myelin sheath covering the neurons present in the central nervous system is degraded result in “degradation of action potential” to reach motor neurons. The reflex would operate if different parts of the circuit were without myelin, there will be decreased impulse conduction or loss of impulse conduction in that sensory-motor area. Depend upon the affected area, the corresponding organs may suffer from paralysis because loss of neurons communication specific regions.

 What is the main cell structure that is affected by multiple sclerosis and how is the action potential of a neuron affected by this disease What is the main ce

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