The neurologist continued In many cases of myasthenia gravis

The neurologist continued; \'In many cases of myasthenia gravis, the antibodies compromise communication from your nerves to your muscles by attacking the receptors for acctylcholinase. When they aren\'t enough functional receptors, the Acetylcholine released onto your muscles can\'t be detected. The edrophonium chloride helped increase the chance that Acetylcholine bound a receptor by reducing the Ach-asc enzyme action, thereby increasing the concentration in the synapse. One good piece of news is that the computer tomography (CT) scan shows that there Is a tumor on your thymus gland- This tumor could be the reason for your problems. The thymus can be easily removed, and this will probably decrease the symptoms; It may even cure them. Additional treatment will probably be necessary to counteract the compromised situation between your nerves and muscles, and it may have to be modified depending on the results of the thyrectomy. What is the general role of the thymus In the body? If you don\'t already know, make some suggestions, according to what you just learned about how Annie\'s may be dysfunctional. Neostigmine is one drug that may be prescribed for people with myasthenia gravis. Go back to the flow diagram, look at each stage, and suggest some possible ways that this drug could work to increase synaptic performance. (It\'s ok if It\'s not the actual answer, but go through the thought process and make some suggestions.)

Solution

2. The general role of thymus gland is to produce various hormones for endocrine gland and also it it is majorly involved in immune system via maturation of T cells. Due to auto immunity there will be over production of T cells which has lead to cancer in the thymus. Removal of thymus will decrease the symptoms, but the person will be susceptible to infections. Instead of that immuno suppressants can be given to suppress the immunological reaction.

3. Neostigmine improves muscle tone in case of myasthenia gravis, and also to reverse the effects of non-depolarizing muscle relaxants such as rocuronium and vecuronium.